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also, the reduction of extracellular potassium increases the degree of inhibition that the drug induces on the rapid potassium current, consequently increasing the QT interval 24

Conversely, despite the promising clinical use of colchicine to prevent inflammasome activation, we observed a significant increase in the IL-1 levels released by patient-derived PBMCs in vitro upon colchicine treatment ( Figure 3E )

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This effect is a milder form of cytolysis compared to Kybellas potent, direct action